SELANK protocol

Dose ranges reflect commonly studied amounts in published research models. This is not medical advice. For research use only. Not for human consumption.

Protocol Comparison

Research Dose Reference

Available in 10 mg

SELANK

Cognitive

Research dose range

0 mcg125 mcg250 mcg375 mcg500 mcg

Standard research range

200 - 350 mcg

Standard research concentration for active stress attenuation and mental stability.

Research focus

Non-sedating emotional stability & GABA balance

Observed window

14-21 days active cycle

Administration

1-2x daily laboratory protocol

Primary pathway

Tuftsin analog / GABA-A modulation / Enkephalin protection

Dosing referenced from published peer-reviewed Russian clinical literature. For laboratory research only.

PROTOCOL BENEFITS

Engineered Outcomes

GABA-A ModulationNon-Addictive AnxiolyticTuftsin Analog

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Synthetic analog of tuftsin providing profound, non-sedating anxiety and stress reduction.

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Promotes positive GABA-A receptor modulation without tolerance, lethargy, or dependence.

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Protects enkephalins from enzymatic breakdown, supporting natural mood and emotional stability.

SCIENCE

How It Works.

Selank is a heptapeptide analog of the endogenous tetrapeptide tuftsin, developed to produce sustained anxiolytic and cognitive-enhancing actions. By binding to allosteric sites on GABA-A receptors, it modulates chloride ion conductance naturally.

In addition, Selank inhibits enkephalin-degrading enzymes in blood plasma, prolonging the half-life of endogenous opioid peptides that regulate emotional equilibrium, resulting in calmness without cognitive blunting or sedation.

WHAT TO EXPECT

Anxiolytic and stress-moderating effects are noticeable within 15–45 minutes. Emotional balance and memory recall under stress are assessed over 2–3 week periods.

PROTOCOL NOTE

Available in 10 mg sterile vials. For laboratory research only.

FAQ

Protocol Questions

What differentiates Selank's mechanism from conventional anxiolytics?+

Selank acts as an allosteric modulator of GABA-A receptors and inhibits enkephalin-degrading enzymes, promoting calmness and focus without sedation, motor impairment, or dependence.

What clinical evidence exists for Selank?+

Clinical trials conducted by the Russian Academy of Sciences demonstrate consistent anxiolytic efficacy in generalized anxiety and neurasthenia cohorts with an excellent tolerability profile.